Refractive, if you want to have an intelligent debate, how about lose the talk.origins response handbook.
Never been there, never heard of it.
Am I going to have to deal with constant nit-picks from you and put you on ignore, or is it possible we can try to have a civil debate?
Fred
So, now you want to rewrite history and cast me as the bad guy?
Huh
Well:
post #23 You say "evos" assume chimp/human ancestry.
post # 25 I inform you that's incorrect
Post # 26 I post a trait and a link.
Post from you to me:
I'm delighted to hear you are the self-proclaimed expert!
I just wish you were an expert in English reading skills.
I have plenty of experience in this debate and can provide articles I have written over the years that prove that I am well aware that evos don't claim we evolved from a monkey. Evos do think however that we evolved from a pile of goo (that's what I would call the first single-celled organism).
Fred
Insults are insults no matter how many emoticons you type in. So let's not think there's going to be some thread mythology promoted about how poor you are being harassed by mean me. You were free to keep the conversation objective at all times.
As to having intelligent debate, I was wondering if you'd like to explain to everyone how a single cell constitutes a "pile."
Also, you seem to think that the difference between monkeys and apes is not significant enough for you to consistently use correct references. As you have been doing this for such a long time, can you explain why you use the terms inconsistently?
Are you claiming that one of Adam's kids could not have been born with a genetic defect that caused him to be sterile?
Be clear, if I decide to stay in this thread, I am fine with going back again and again and again every time you try to divert and reposting your original questions. I will paraphrase once unless you try to deny what you asked: You said that you wanted an example of a trait the could not have come from Adam and Eve's genome.
Genetically derived sterility is one. Not disease-derived sterility which is an acquired trait.
You asked for a trait found in humans that could not come from the genomes of Adam or Eve. My question is: besides this one, how many would you like?
Genetics of human male infertility.
Poongothai J, Gopenath TS, Manonayaki S.
Source
Computational Engineering and Networking Department, Amrita Viswha Vidyapeetham, Ettimadai, Coimbatore 638107, Tamil Nadu, India.
poongothai_jp@yahoo.co.in
Abstract
Infertility is defined as a failure to conceive in a couple trying to reproduce for a period of two years without conception. Approximately 15 percent of couples are infertile, and among these couples, male factor infertility accounts for approximately 50 percent of causes. Male infertility is a multifactorial syndrome encompassing a wide variety of disorders. In more than half of infertile men, the cause of their infertility is unknown (idiopathic) and could be congenital or acquired. Infertility in men can be diagnosed initially by semen analysis. Seminograms of infertile men may reveal many abnormal conditions, which include azoospermia, oligozoospermia, teratozoospermia, asthenozoospermia, necrospermia and pyospermia. The current estimate is that about 30 percent of men seeking help at the infertility clinic are found to have oligozoospermia or azoospermia of unknown aetiology. Therefore, there is a need to find the cause of infertility.
The causes are known in less than half of these cases, out of which genetic or inherited disease and specific abnormalities in the Y chromosome are major factors. About 10-20 percent of males presenting without sperm in the ejaculate carry a deletion of the Y chromosome. This deleted region includes the Azoospermia Factor (AZF) locus, located in the Yq11, which is divided into four recurrently deleted non-overlapping subregions designated as AZFa, AZFb, AZFc and AZFd. Each of these regions may be associated with a particular testicular histology, and several candidate genes have been found within these regions. The Deleted in Azoospermia (DAZ) gene family is reported to be the most frequently deleted AZF candidate gene and is located in the AZFc region. Recently, a partial, novel Y chromosome 1.6-Mb deletion, designated "gr/gr" deletion, has been described specifically in infertile men with varying degrees of spermatogenic failure. The DAZ gene has an autosomal homologue, DAZL (DAZ-Like), on the short arm of the chromosome 3 (3p24) and it is possible that a defective autosomal DAZL may be responsible for the spermatogenic defect. The genetic complexity of the AZF locus on the long arm of the Y chromosome could be revealed only with the development of sequence tagged sites. Random attacks on the naked mitochondrial DNA (mtDNA) of sperm by reactive oxygen species or free radicals will inevitably cause oxidative damage or mutation to the mitochondrial genome with pathological consequences and lead to infertility in males. The key nuclear enzyme involved in the elongation and repair of mtDNA strands is DNA polymerase gamma, mapped to the long arm of chromosome 15 (15q25), and includes a CAG repeat region. Its mutation affects the adenosine triphosphate production. The introduction of molecular techniques has provided great insight into the genetics of infertility. Yet, our understanding of the genetic causes of male infertility remains limited.
